Relationship Between Job Strain and Myocardial Infarction in The National Cardiovascular Center Patients

Rima Melati; Endang Basuki; Budhi Setianto

doi:10.30701/ijc.v29i1.197

Rima Melati Departemen Ilmu Kedokteran Komunitas, Program Studi Magister Kedokteran Kerja, Fakultas Kedokteran Universitas Indonesia
Endang Basuki Kepala Sub Departemen Manajemen Kedokteran. Departemen Ilmu Kedokteran Komunitas, Fakultas Kedokteran Universitas Indonesia
Budhi Setianto Departemen Kardiologi dan Kedokteran Vaskular, Fakultas Kedokteran Universitas Indonesia, Rumah Sakit Jantung dan Pembuluh Darah Harapan Kita – Pusat Jantung Nasional Harapan Kita, Jakarta

DOI: https://doi.org/10.30701/ijc.v29i1.197

Abstract

Background. Coronary heart disease is the most frightening disease and still become a problem in the developed and developing countries. The prevalence of myocardial infarction is also increasing from year to year. Beside the conventional risk factors, it is also influenced by occupational factors. Although job strain can cause stress which would have impact on the occurence of myocardial infarction, the prevention strategies being implemented are just for conventional risk factors. There is still no concern for occupational factors which can also cause job strain. This study was aimed to assess the relationship between job strain and other risk factors with myocardial infarction among workers.
Methods. The study design was matched case – control 1:1 for age. Data were collected by using general questionnaire which covered demography characteristics, conventional risk factors, job characteristics, and demand – control questionnaire (JCQ) to assess job strain.
Result. Job strain, smoking and dyslipidemia were risk factors which had relationship with myocardial infarction. Job strain increased myocardial infarction risk by 6.8 times (Adj OR 6.80, 95% CI: 2.72 ; 16.98, p = 0.000). Light smokers increased myocardial infarction risk by 15 times (Adj OR 14.97, 95% CI: 3.17 ; 70.74, p = 0.001), medium smokers increased myocardial infarction risk by 7.7 times (Adj OR 7.72, 95% CI: 2.73 ; 21.84, p = 0.000), and heavy smokers increased myocardial infarction risk by 26 times (Adj OR 25.61, 95% CI: 5.25 ; 124.88, p = 0.000). Dyslipidemia increased myocardial infarction risk by 2.8 times (Adj OR 2.82, 95% CI: 1.07 ; 7.44, p = 0.035). Job strain component which increased myocardial infarction risk was high job demand (Adj OR 2.44, 95% CI: 1.02 ; 5.85, p = 0.046).
Conclusion. Job strain, smoking and dyslipidemia simultaneously had relationship with myocardial infarction.

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References

Majid A. Penyakit jantung koroner: patofisiologi, pencegahan, dan pengobatan terkini. Available from: http://www.usu.ac.id/id/files/pidato/ppgb/2007.

Kusmana D et al. Jakarta cardiovascular study report 1. Rumah sakit jantung dan pembuluh darah Harapan Kita. Jakarta. 2008.

Gaziano JM, Manson JE, Ridker PM. Primary and secondary prevention of coronary heart disease. In: Libby P et al, editors. Braunwald’s heart disease: a textbook of cardiovascular medicine. 8th ed. Philadelphia: Saunders Elsevier; 2008. 1119 – 48.

Yusuf S, Hawken S, Ounpuu S, Dans T, Avezum A, Lanas F et al. Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries (the INTERHEART study): case-control study. Lancet. 2004;364: 937 – 52.

Yusuf S, Hawken S, Ounpuu S, Bautista L, Franzosi MG, Commerford P et al. Obesity and the risk of myocardial infarction in 27000 participants from 52 countries: a case-control study. Lancet. 2005;366:1640 – 49.

Pelfren E, De Backer G, Mak R, de Smet P. Kornitzer M. Job stress and cardiovascular risk factors. Results from BELSTRESS study. Arch Public Health. 2002; 60: 245 – 68.

Malinauskien? V, Azaraviciene A, Apelis V. The demand – control model and myocardial infarction in the working population of Kaunas men. Acta Medica Lituanica. 2004;11( 4): 32 –5.

Netterstrom B, Nielsen FE, Kristensen TS, Bach E, Moller L. Relation between job strain and myocardial infarction: a case-control study. J Occup Environ Med 1999; 56: 339-42.

Éboulé CA, Brisson C, Maunsell E, Mâsse B, Bourbonnais R, Vézina M et al. Job strain and risk of acute recurrent coronary heart disease events. JAMA. 2007;298(14):1652 –60.

Cahill J, Landsbergis PA, Schnall PL. Reducing occupational stress. Presented at the work stress and health conference. Washington DC. 1995 September.

Houtman I, Kornitzer M, de Smet P, Koyuncu R, de Backer G, Pelfrene E. Job stress, absenteeism and coronary heart disease European cooperative study (the JACE study). Eur J Public Health. 1999;9(1): 52 – 7.

Theorell T, Tsutsumi A, Hallquist J, Reuterwall C, Hogstedt C, Fredlund P et al. Decision latitude, job strain, and myocardial infarction: a study of working men in Stockholm. Am J Public Health. 1998;88:382 – 8.

Hammar N, Alfredsson L, Johnson JV. Job strain, social support at work, and incidence of myocardial infarction. Occup Environ Med. 1998;55:548 - 53

Omvik P. How smoking affect blood pressure. Rev Blood Press. 1996;5:71 – 7.

Aveyard P, West R. Managing smoking cessation. BMJ. 2007 July 7;335:37 – 41.

Hennrikus DJ, Jeffrey RW, Lando HA, Murray DA, Brelje K, Davidann B et al. The SUCCESS project: The effect of program format and incentives on participation and cessation in worksite smoking cessation programs. Am J Public Health. 2002;92:274 – 9.